Editing 1640: Super Bowl Context
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Cueball feels the need to disseminate any information he finds interesting, even in trivial conversation. Normally people like to have context-free conversations{{Citation needed}} and White Hat invites Cueball to try to fit in with normal people's conversational style. | Cueball feels the need to disseminate any information he finds interesting, even in trivial conversation. Normally people like to have context-free conversations{{Citation needed}} and White Hat invites Cueball to try to fit in with normal people's conversational style. | ||
โ | Cueball apologizes and agrees to try, but even though he really tries hard, with | + | Cueball apologizes and agrees to try, but even though he really tries hard, with White Hat encouraging him to just reply normally to a question about the [http://profootballtalk.nbcsports.com/2016/02/01/report-peyton-manning-has-told-close-friends-hell-retire/ rumored retirement] of {{w|Peyton Manning}}, he cannot stop himself from including context in his reply again. White Hat probably wanted Cueball to join in such minimal-context speculation. But, failing miserably again, White Hat finally gives up, and suggests they should try another conversation in a year, when Cueball might have learned to talk about the Super Bowl without context (hence the title). |
This time he goes off on a tangent about Peyton as a {{w|mammal}}, and then adding the process of {{w|aging}} and mentioning two reasons for this (which are not well understood). The first he mentions is {{w|Ageing#Damage-related factors|accumulation of damage}}, which includes {{w|mutations}} that can lead to diseases such as {{w|cancer}}. The other process he mentions is {{w|Ageing#Programmed_factors|timed factors}} which includes {{w|telomeres}}. These have been linked to {{w|senescence|biological aging}} because of the shortening of telomeres at each {{w|Cell cycle|cell division}}; when telomeres become too short, the cells die (and so do mammals). | This time he goes off on a tangent about Peyton as a {{w|mammal}}, and then adding the process of {{w|aging}} and mentioning two reasons for this (which are not well understood). The first he mentions is {{w|Ageing#Damage-related factors|accumulation of damage}}, which includes {{w|mutations}} that can lead to diseases such as {{w|cancer}}. The other process he mentions is {{w|Ageing#Programmed_factors|timed factors}} which includes {{w|telomeres}}. These have been linked to {{w|senescence|biological aging}} because of the shortening of telomeres at each {{w|Cell cycle|cell division}}; when telomeres become too short, the cells die (and so do mammals). |